Chad Fabry

Download Attachment: tiedup.jpg 53.74 KB The same guy who lied about his furnace, water heater, and roof also lied about his electrician.

Thanks guys. I just reported the seller was a dirty rotten liar and both units were undersized, old and ugly. I also went on to state that the install was crummy as well and it would cost a lot to fix it all. I appreciate the help and dates, Brian et al

no rush, I'm writining the report now and I can't go home untill someone answers. Bryant 50k btu furnace.394GAW024050/0981C76101 Reliance water heater 30k btu K96888704 The owners say the furnace is 5 years old...I'd say closer to 25 and they say the water heater is 3 years old, I'm guessing 10

Don, My father owns a military aircraft deicing truck w/ an 80 ft man boom they used to apply the glycol solutions. Even in a boom bucket, 80 feet is wobbly and scary as hell.

I hate 40ft ladders. I hate climbing them even more.

Sorry, it's a 200 amp panel with 40 breakers, and a 100 amp se cable. The panel is fused at 100 amps.

Is it allowed to have a 200 amp panel with 40 circuits and only have 100 amp mains? I can't think of any reason it'd be a problem.

You could come and help me put the engine in this Chevy Lumina APV van..I guarantee you'll want to be a home inspector.

I'm not advocating these, but I used some on a friend's house. ( he bought the supplies ) They were handy, I admit it, but I don't think I'd use them at my house. I like twisting and wire nuts. Anyway,free is free. http://www.idealindustries.com/wt/insur ... n?OpenForm

I was kidding about the carpet. I couldn't find eight things.

uh, that book's a hundred dollars. I have to do some odds figurin here. 12 stucco homes in my area of four hundred thousand, someone has to sell or buy a stucco home. They have to hire a home inspector. They have to somehow find my name even though I'm not yet in the phone book. They have to feel confident enough to hire a guy that's done 8 home inspections total, and they have to get past the fact that I gasped when I heard it was stucco.

I want that manual. C'mon, share w/ the new guy.

That wouldn't last long around here, pressure treated or not. To this day I can't imagine what the settlers were thinking when this area was first inhabited.

What the heck, I'll give it a go. Spindle spacing too wide. Gate doesn't close and latch by itself, walkway is too narrow under that ugly bay window or shrine or whatever that is. The light fixture is too low, the vent is discharging on top of that bay thing and also into the pool area, the light switch for the light that's too low seems to not be weatherproof, and I don't see any bonding on any of the window frames that look like aluminum, or the the vent thing. I'm sure that given the level of craftsmanship, they've just cleverly concealed the bonding. Lastly, the illegal use of indoor / outdoor carpet.

By the Grace of God it was the second link that fixed my problem. regsvr32 urlmon.dll somethin, somethin, somethin, gizmo was out of whack. Thanks for all the help

Well then, I guess that rules out spelunking for asthmatics. And birds. And farming. And working in plastics plants. And household dust. And pets. Since grain farmers and poultry raisers have the highest incidence of the disease, these industries should be eliminated or highly regulated. I think it's best to eliminate conditions that encourage mold, and with rare exception (bleu cheese)I'm not a proponent of mold. That said, I think the whole mold scare is dramatically overexposed and I'd be willing to bet that no more than one percent of lawsuits are justified. Background: Hypersensitivity pneumonitis (HP), also called extrinsic allergic alveolitis, is a complex syndrome of varying intensity, clinical presentation, and natural history, rather than a single uniform disease. First described in Iceland in 1874 and termed heykatarr, HP is caused by sensitization to repeated inhalation of dusts containing organic antigens. These dusts can be derived from a variety of sources, such as dairy and grain products, animal dander and protein, wood bark, and water reservoir vaporizers. The most common antigens are thermophilic actinomycetes and avian proteins; the most common diseases are farmer's lung and bird fancier's lung. HP is characterized by diffuse inflammation of lung parenchyma and airways in previously sensitized patients. Based on the length and intensity of exposure and subsequent duration of illness, clinical presentations of HP are categorized as acute, subacute (intermittent), and chronic progressive. Pathophysiology: Pathologically, acute HP is characterized by poorly formed noncaseating interstitial granulomas and mononuclear cell infiltration in a peribronchial distribution with prominent giant cells. The subacute, or intermittent, form produces more well-formed noncaseating granulomas, bronchiolitis with or without organizing pneumonia, and interstitial fibrosis. Chronic forms reveal additional findings of chronic interstitial inflammation and alveolar destruction (honeycombing) associated with dense fibrosis. Cholesterol clefts or asteroid bodies are present within or outside granulomas. Pathogenesis Most patients have circulating immunoglobulin G antibodies that are specific for the offending antigen. The antibody (called precipitating antibody) reacts with a specific antigen to form a precipitation. However, approximately 50% of asymptomatic persons exposed to the sensitizing antigen also have these antibodies. Although initially thought to be an immunocomplex-mediated process, later studies showed that cell-mediated immunity is more important. Early response to the antigen is characterized by an increase in neutrophils in the alveoli and small airways followed by an influx of mononuclear cells. These cells release proteolytic enzymes, prostaglandins, and leukotrienes. The production and release of interleukins, cytokines, growth factors, and various other mediators from T lymphocytes and macrophages play important roles in HP pathogenesis. Frequency: In the US: Resistance or susceptibility to infection following exposure varies. Incidence also varies considerably. Studies document 8-540 cases per 100,000 persons per year for farmers and 6000-21,000 cases per 100,000 persons per year for pigeon breeders. High attack rates are documented in sporadic outbreaks. Approximately 52% of office workers exposed to an infected humidifier were infected, and 27% of workers at a molding plant for polyurethane foam parts were infected. Prevalence varies by region, climate, and farming practices. HP affects 0.4-7% of the farming population. Reported prevalence among bird fanciers is estimated to be 20-20,000 cases per 100,000 persons at risk. Internationally: The prevalence of farmer's lung in the United Kingdom is reported to be 420-3000 cases per 100,000 persons at risk, in France is 4370 cases per 100,000 persons at risk, and in Finland is 1400-1700 cases per 100,000 persons at risk. Mortality/Morbidity: Most patients recover completely after the inciting exposure ceases. Bird fancier's disease has a worse prognosis than farmer's lung. The outcomes of other varieties of HP are more variable. CLINICAL Section 3 of 11 Author Information Introduction Clinical Differentials Workup Treatment Medication Follow-up Miscellaneous Pictures Bibliography History: The clinical presentation of HP is categorized as acute, subacute, or chronic, according to duration of illness. Acute HP The acute form may develop 4-6 hours following heavy exposure to an inciting agent. Symptoms often resolve spontaneously within 12 hours to several days upon cessation of exposure. Patients abruptly develop fever, chills, malaise, cough, chest tightness, dyspnea, headache, and malaise. Subacute (intermittent) HP Patients may gradually develop a productive cough, dyspnea, fatigue, anorexia, and weight loss. Findings may be present in patients who experience repeated acute attacks. Chronic HP Patients often lack a history of acute episodes. They have an insidious onset of cough, progressive dyspnea, fatigue, and weight loss. Removing exposure results in only partial improvement. Physical: Physical examination findings vary according to clinical presentation. Patients with acute HP present with fever, tachypnea, and diffuse fine bibasilar crackles upon auscultation. Patients with subacute HP present similarly to patients with acute disease, but symptoms are less severe and last longer. Patients with chronic HP present with muscle wasting and weight loss. Clubbing is observed in 50% of patients. Tachypnea, respiratory distress, and inspiratory crackles over lower lung fields often are present. Causes: More than 300 etiologies of HP have been reported from a wide range of exposures involving airborne antigens. Selected Etiological Agents for HPDisease Source of Exposure Major Antigen Farmer's lung Moldy hay Saccharopolyspora rectivirgula (Micropolyspora faeni) Bagassosis Moldy sugar cane fiber Thermoactinomyces sacchari Grain handler's lung Moldy grain S rectivirgula, Thermoactinomyces vulgaris Humidifier/air-conditioner lung Contaminated forced-air systems, heated water reservoirs S rectivirgula, T vulgaris Bird breeder's lung Pigeons, parakeets, fowl, rodents Avian or animal proteins Cheese worker's lung Cheese mold Penicillium casei Malt worker's lung Moldy malt Aspergillus clavatus Paprika splitter's lung Paprika dust Mucor stolonifer Wheat weevil Infested wheat Sitophilus granarius Mollusk shell hypersensitivity Shell dust Sea snail shells Chemical worker's lung Manufacture of plastics, polyurethane foam, rubber Trimellitic anhydride, diisocyanate, methylene diisocyanate Reported occupations and major causative antigens are as follows: Farmers and cattle workers develop the most common form of HP. The major causative antigen is thermophilic actinomycetes. Farmer's lung must be distinguished from febrile toxic reactions to inhaled mold dusts (organic dust toxic syndrome). This nonimmunologic reaction occurs 30-50 times more commonly than HP. Ventilation workers and those exposed to water-related contamination may be exposed to microorganism-colonized forced-air systems, humidifiers, whirlpools, hot tubs, and spas. Antigens are various species of Thermoactinomyces or Cladosporium. Poultry and other bird handlers are commonly exposed to droppings, feathers, and serum proteins of pigeons, other birds, and fowl Veterinarians and animal handlers have significant contact with animals and organic antigens. Grain and flour processors and loaders are exposed to grain that may become colonized with a variety of microorganisms that are easily aerosolized. Exposure may lead to HP. Lumber mill workers and paper and wallboard manufacturers are exposed to wood products colonized with molds. Plastic manufacturers, painters, and electronics industry workers may be exposed to inciting agents that are synthetic in origin, possibly including diphenylmethane diisocyanate or toluene diisocyanate. Textile workers may have exposures that lead to lung injury characterized by diffuse alveolar damage or airway dysfunction (eg, byssinosis, nylon worker's lung). This is not a true form of HP. Conditions that mimic HP that occur from inhalation of organic agents but are not true forms of HP are as follows: Patients with inhalation fever present with fever, chills, headaches, and myalgias without pulmonary findings (although mild dyspnea may occur). Onset is 4-8 hours following exposure, but no long-term sequelae occur. Organic dust toxic syndrome results from exposure to bioaerosols contaminated with toxin-producing fungi (mycotoxins). Fever, chills, and myalgias occur 4-6 hours after exposure, and chest radiographs may show diffuse opacities. Bronchiolitis or diffuse alveolar damage may be present on lung biopsy specimens. These are not true forms of HP because no prior sensitization is required. Chronic bronchitis can result from chronic obstructive pulmonary disease, which is the most common respiratory syndrome among agricultural workers. The prevalence of chronic bronchitis is 10%, compared with 1.4% for HP. Smoking and atopy have additive effects. An association may exist between chronic bronchitis and HP.

Hi Laufen, It needs to be fixed, the only question is who's paying to fix it.(Damn I ended another sentence w/ a preposition.)

It's your house. Sell it or not, the choice is yours. The number doesn't seem terribly unreasonable to install vents and do some kind of clean up. Personally, I'd tell 'em to rub rocks. But that's just me. It all depends on poker and who has the cards in the deal.

The hose photo sticks out to me because of the efflorescence on the patio under the hose path and then continuing to the edge. The hose bib connection probably drips a little and follows the hose to the concrete. This indicates the hose is on often and for a long time. I don't get the screen enclosure failure other than the fact it's broken. The electrical double tap is obvious, but for the life of me I can't figure out why they've double tapped unless the spa isn't fused at all. I'm assuming that the sub panel is the feeder for the pool and the spa.

I'm using outlook express as my mail program with XP pro. When I get mail w/ a link in the text it appears properly as a hyperlink but doesn't work when I click on it. I have to copy the link and paste it to the search window. Anyone know how to fix that?

I think so too.

The pressure preset in the tank is too high. The air pressure in the empty tank with a 30/50 switch should be about 27 lbs. That'll leave some water in the tank before the pump kicks in at 30lbs. There's a schrader tire style valve at the top of the tank where you can adjust the pressure.

I'm bettin the kid now has to deal with an unheated pool. Oh, the woes of the working class. Now, I think I only had one of the four problems identified correctly. That's exactly why I didn't expose my soft white underbelly on the earlier pool questions. Give us more Norm; being wrong only sucks about half as bad as I thought it would.

Does it have something to do with the fact that the returns are directly opposite the intake and the floating chlorinator probably hovers in the same spot all the time?

1)I'm guessing the bubbles are from a leak in the pump suction line, or the water level is too low and air is entering the skimmer. 2)The lights will overheat and break the lenses if not fully submerged? 3)I don't know what the photo is w/ the clamp light in it except I can see heavily corroded galvanized pipe. Chlorine is a potent oxidizer and would greatly accelerate the deterioration of exposed metal. 4) I have no idea what caused the dark stain. My first guess would be that area is protected from sunlight and the rest of the liner faded.